PEP/NEWS — Peptide intel. Daily.

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Morning Erections. How would you feel if you were not getting the morning wood?

What happens when you quit social media for seven days? In this video, I designed a simple seven-day protocol to observe the results and by the end of the week, I uncovered something hiding in plain sight that changed my life. 🥦 Start you

Researchers used aging trajectories of individual cell types to predict diseases like Alzheimer's and lung cancer. The finding strengthens the case for tissue-specific aging markers over single-clock approaches.

Researchers found a primate-specific non-coding RNA strand that makes cellular senescence worse. Non-coding RNAs don't build proteins, but they regulate gene expression. This one appears to push aging forward.

Fight Aging! compiled a reference list of interventions with human evidence for reducing epigenetic age — the biological clock read from DNA methylation patterns. Useful for anyone benchmarking protocols against the literature.

NeuroAge Therapeutics opened registration for Younger 2027, a six-month competition scored on clinical-grade aging panels. Baseline kits ship in September. The format creates a real-world dataset for biological age interventions.

Too much HIF-1α (a protein that responds to low oxygen) triggers unwanted blood vessel growth in cartilage, destroying the tissue and causing osteoarthritis. The finding maps a pathway relevant to peptide-mediated joint research.

A 466-patient cluster analysis found that glutathione (a key antioxidant) supplementation benefits patients with advanced, unstable liver disease — but not stable cirrhosis. Patient selection, it turns out, is everything.

Bryan Johnson's Blueprint team published the four cardiovascular protocols behind his vascular age measurements. Blood pressure lower than 90% of 18-year-olds. Whether you buy his methods or not, the biomarker data is documented.

A systematic review found that randomized trial data and real-world observational data disagree on whether anti-obesity medications raise cognitive disorder risk. The discrepancy matters for anyone tracking long-term GLP-1 outcomes.

Protein-based aging clocks predicted chronic disease onset and mortality better than chronological age across two large European cohorts. The data strengthens the case for proteomics as a practical readout of biological aging.

Blocking the damage-signaling response at shortened telomeres — rather than lengthening telomeres directly — restored blood cell production and immune function in preclinical models. A distinct angle on telomere-targeted aging research.

Researchers used drug-disease network mapping to find existing approved drugs that target multiple aging hallmarks at once. Several compounds with peptide-adjacent mechanisms appeared in the output.