PEP/NEWS — Peptide intel. Daily.

Understanding how your home environment shapes your sexual development is key to healthy relationships. In this episode, Dr. Debra Durst sits down with New York Times best-selling author and AASECT-certified sex therapist Ian Kerner to unp

Lifespan.io's monthly digest covers July's standout longevity research, clinical updates, and one serious setback in the field. A fast read for anyone tracking the state of rejuvenation medicine.

Research published in Aging Cell shows that neutrophil extracellular traps (NETs) worsen UVB-induced skin damage. Inhibiting NETs reduced that damage in the study model — relevant context for anyone following photoaging and skin repair peptide research.

Human Longevity Inc.'s CEO makes the case for individual health ownership and data-driven longevity. The interview covers where the company stands after a turbulent decade and what its genomic-and-phenotypic approach adds to the field.

Kate Tolo documents how she reversed measurable skin age markers using multispectral imaging to track 8 biomarkers over time. The piece details every intervention she used — a useful reference for researchers following skin-repair and photoaging endpoints.

Reactive medicine waits for organ failure. This piece argues that the economic cost of that model — in lost productivity, chronic disease burden, and downstream care — makes equitable access to longevity tools a fiscal necessity.

Methylation-based biological age clocks are generally consistent when inputs are stable — but short-term stress events can swing results dramatically. Researchers say this is a key confound when using clocks to evaluate intervention response.

Dr. José Pedro Castro, formerly of the Gladyshev Lab, discusses how chronic low-grade inflammation accelerates biological aging and what the research says about slowing it. Directly relevant to anti-inflammatory peptide research.

Researchers found that blocking SLC25A1 — a mitochondrial citrate transporter — cuts the inflammatory secretions that senescent cells use to disrupt surrounding tissue. A potential new handle for senolytic and anti-inflammaging strategies.